What is the Benefits of Marijuana and its Risk?


Marijuana is a green, brown, or gray mixture of dried, shredded leaves, stems, seeds, and flowers of the hemp plant. You may hear marijuana called by street names such as pot, herb, weed, grass, boom, Mary Jane, gangster, or chronic. There are more than 200 slang terms for marijuana.

Marijuana Dangers

* Impaired perception
* Diminished short-term memory
* Loss of concentration and coordination
* Impaired judgement
* Increased risk of accidents
* Loss of motivation
* Diminished inhibitions
* Increased heart rate
* Anxiety, panic attacks, and paranoia
* Hallucinations
* Damage to the respiratory, reproductive, and immune systems
* Increased risk of cancer
* Psychological dependency

Marijuana as Medicine

Although U.S. law classifies marijuana as a Schedule I controlled substance (which means it has no acceptable medical use), a number of patients claim that smoking pot has helped them deal with pain or relieved the symptoms of glaucoma, the loss of appetite that accompanies AIDS, or nausea caused by cancer chemotherapy. There is, however, no solid evidence that smoking marijuana creates any greater benefits than approved medications (including oral THC) now used to treat these patients, relieve their suffering, or mitigate the side effects of their treatment. Anecdotal assertions of beneficial effects have yet to be confirmed by controlled scientific research.

Health benefits are well documented, from depression and anxiety relief to reduced blood pressure, pain alleviation and glaucoma treatment. It is not addictive, does not kill brain cells and is not a “gateway” drug - in fact, when pot is more available, studies show that the use of hard drugs like heroin and cocaine actually decreases. The bottom line for hikers: when your leg is broken from a misjudged boulder hopping attempt (pain) and a bear has eaten your friend (depression) and you’re lost because you forgot the compass (dumbass), consult the cannabis.

Acute effects of smoked marijuana and oral delta-9-tetrahydrocannabinol on specific airway conductance in asthmatic subjects

The acute effects of smoked 2 per cent natural marijuana (7 mg per kg) and 15 mg of oral delta-9-tetrahydrocannabinol (THC) on plethysmographically determined airway resistance (Raw) and specific airway conductance (SGaw) were compared with those of placebo in 10 subjects with stable bronchial asthma using a double-blind crossover technique. After smoked marijuana, SGaw increased immediately and remained significantly elevated (33 to 48 per cent above initial control values) for at least 2 hours, whereas Sgaw did not change after placebo. The peak bronchodilator effect of 1,250 mcg of isoproterenol was more pronounced than that of marijuana, but the effect of marijuana lasted longer.

After ingestion of 15 mg of THC, SGaw was elevated significantly at 1 and 2 hours, and Raw was reduced significantly at 1 to 4 hours, whereas no changes were noted after placebo. These findings indicated that in the asthmatic subjects, both smoked marijuana and oral THC caused significant bronchodilation of at least 2 hours' duration.

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